좀 길게 늘어지는 것같아요
그래도 잘봤습니다
여주가 조금만 더 당당했다면 좋을건데..

복습이 쉽지않는 내용이네요
여주인 장공주가 너무여리여리 하다랄까요
수동적이라는말밖에는...아쉬워요

리턴해서 이렇게 되돌릴수 있으면 얼마나 좋을까 라는 생각을 하면서 읽었네요~ 지루하지 않게 읽었습니다~

“주인의 허락도 없이, 어찌 노비가 제 마음대로 아랫도리를 휘두르고 다니겠습니까.”


몰락한 귀족가의 여식 서은린의 꿈은 편찮은 아버지, 집안의 유일한 몸종인 도하와 오손도손 사는 것뿐이다.
어느 날, 황궁 사람들이 집으로 들이닥쳐 황태자비가 되라는 황명을 전하곤 은린을 강제로 끌어낸다.
북궁, 황궁 내에서 가장 어둡고 외진 데 유폐된 황태자의 비가 되라니, 그녀의 앞날은 그 북궁처럼 어둡고 춥기만 할 것이 뻔할 터. 은린은 죽음을 각오하고 제 마음을 따르기로 하는데…….


“저로 인해 여인이 되셨으니, 아기씨께서 저를 사내로 만들어주세요.”
“무…….”
“겨우 그 정도의 자극으로 포궁이 열리지 않았습니까. 아니라고 말하는 작은 입술과는 다르게 아래의 입은 정직해서요.”
은린은 숨을 들이켰다. 달거리 이야기다.
“내가…… 다른 동무들보다 늦은 거다. 너 때문이 아니라 때가 돼서…….”
“무엇이라도 좋습니다. 아기씨께서 저를 사내로만 만들어주신다면.”
작가 소개

따뜻한 글 잘 보았습니다.. 애틋한 마음이 가 더 몰입을 해 읽었네요.. 줄어드는 페이지와 마음에.. 추석 모쪼록 따뜻한 것이 좋은 계절에 따뜻한 글 고맙습니다..건필하세요

ften first noticed because of abdominal swelling or a mass in the kidney that can be felt upon physical examination. Some affected children have abdominal pain, fever, a low number of red blood cells (anemia

Wilms tumor is a form of kidney cancer that primarily develops in children. Nearly all cases of Wilms tumor are diagnosed before the age of 10, with two-thirds being found before age 5.

Wilms tumor is

In most cases, people with MELAS inherit an altered mitochondrial gene from their mother. Less commonly, the disorder results from a new mutation in a mitochondrial gene and occurs in people with no family history of MELAS.

disorders resulting from mtDNA mutations from their mother. These disorders can appear in every generation of a family and can affect both males and females, but fathers do not pass traits

contained in mtDNA. Because egg cells, but not sperm cells, contribute mitochondria to the developing embryo, children can only inherit

This condition is inherited in a mitochondrial pattern, which is also known as maternal inheritance. This pattern of inheritance applies to genes

he ability of mitochondria to make proteins, use oxygen, and produce energy. Researchers have not determined how changes in mtDNA lead to the specific

Mutations in a particular transfer RNA gene, MT-TL1, cause more than 80 percent of all cases of MELAS. These mutations impair the ability

which are chemical cousins of DNA. These molecules help assemble protein building blocks called amino acids into full-length, functioning proteins within mitochondria.

n mitochondria that helps convert oxygen, fats, and simple sugars to energy. Other genes associated with this disorder provide instructions for making molecules called transfer RNAs (tRNAs

Some of the genes related to MELAS provide instructions for making proteins involved in normal mitochondrial function. These proteins are part of a large enzyme complex

food into a form that cells can use. Although most DNA is packaged in chromosomes within the nucleus, mitochondria also have a small amount of their own DNA, known as mitochondrial DNA or mtDNA

MELAS can result from mutations in one of several genes, including MT-ND1, MT-ND5, MT-TH, MT-TL1, and MT-TV. These genes are found in the DNA of cellular structures called mitochondria, which convert the energy from fo

acidosis. Increased acidity in the blood can lead to vomiting, abdominal pain, extreme tiredness (fatigue), muscle weakness, and difficulty breathing. Less commonly, people with MELAS may experience involuntary muscle spasms (myoclonus),

the brain, leading to vision loss, problems with movement, and a loss of intellectual function (dementia).

Most people with MELAS have a buildup of lactic acid in their bodies, a condition called lactic